A corrected clubfoot is often a shoe size smaller than normal and may be somewhat less flexible.
A family history of clubfoot has been reported in 24.4 percent of families in a single study.
A pediatrician should be consulted at birth, the usual time clubfoot is initially diagnosed.
At the present time, there is no way to prevent clubfoot.
Clubfoot can affect one foot or both feet.
Clubfoot is a condition in which one or both feet are twisted into an abnormal position at birth.
Clubfoot is corrected by casting or surgery.
Clubfoot is diagnosed by physician inspection.
Clubfoot may be suspected during the latter stages of pregnancy, especially in a mother of shorter or smaller than normal stature, a large fetus, or multiple infants.
Experts do not agree on the precise cause of clubfoot.
For a person with clubfoot, walking becomes difficult or impossible.
For unknown reasons, amniocentesis, a prenatal test, has also been associated with clubfoot.
However, without treatment, clubfoot will result in a functional disability.
If there is no anatomic abnormality of the bone, this is not true clubfoot, and the problem can usually be corrected by applying special braces or casts to straighten the foot.
In talipes varus, the most common form of clubfoot, the foot generally turns inward so that the leg and foot look somewhat like the letter J (when looking at the left foot head-on).
Intrauterine exposure to the drug misoprostol has been linked with clubfoot.
J., et al. "Leg length discrepancy in unilateral congenital clubfoot following surgical treatment."
Many obstetricians feel that intrauterine crowding causes clubfoot.
Orthopedic surgery may be necessary for congenital clubfoot deformity.
Parents of an infant with clubfoot should be prepared to participate in treatment for two or more years.
Persons with clubfoot that is corrected by surgery may notice some increased stiffness in their affected feet as they age.
Pregnant women can reduce the risk of clubfoot by refraining from smoking.
Researchers in Norway have reported that males who are in the printing trades have significantly more offspring with clubfoot than men in other occupations.
Several environmental causes have been proposed for clubfoot.
Skeletal abnormalities may include poorly developed shoulder blades (scapulae), sideways bent fingers (clinodactyly), clubfoot, scoliosis, and unusual neck bones.
Some experts feel that clubfoot may begin early in pregnancy, probably in the 10th to 12th weeks of gestation.
The calf muscles in an affected clubfoot leg may be slightly smaller than an unaffected leg.
The focus will be on treating clubfoot but other lower limb conditions and general issues are covered.
The infants of mothers who smoke during pregnancy have a greater chance of being born with clubfoot than are offspring of women who do not smoke.
The MSX1 gene has been associated with clubfoot in animal studies.
The physical appearance of a clubfoot may vary.
The prognosis for successfully treating clubfoot is good at this time.
The ratio of males to females with clubfoot is 2.5 to 1.
There may also be some hip bone anomaly or other skeletal symptoms, for example, clubfoot.
There was a lot of debate over whether the method was really suitable for severe intractable clubfoot especially over the correction of tibial torsion.
These findings suggest the potential role of one or more genes being responsible for clubfoot.
This theory is supported by a significantly higher incidence of clubfoot among twins compared to singleton births.
True clubfoot is characterized by abnormal bone formation in the foot.
True clubfoot is usually obvious at birth because a clubfoot has a typical appearance of pointing downward and being twisted inwards.
Uncorrected clubfoot in an adult causes only part of the foot, usually the outer edge or the heel or the toes, to touch the ground.