keratinocytes in A Sentence

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    The migration of endothelial cells and Keratinocytes.

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    Keratinocytes migrate without first proliferating.

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    Before they begin migrating, Keratinocytes change shape, becoming longer

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    Or 2 protect Keratinocytes from the deleterious effects of primary antibody binding.

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    Of these, Keratinocytes are the major component, constituting roughly 95 percent of the epidermis.

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    TB-500 is different from other repair factors, because it promotes endothelial and keratinocyte migration.

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    This process happens via Keratinocytes which grow in the lower layers of the epidermis, starting at the stratum basale;

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    Migration of Keratinocytes over the wound site is stimulated by lack of contact inhibition and by chemicals such as nitric oxide.

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    Before they begin migrating, Keratinocytes change shape, becoming longer and flatter and extending cellular processes like lamellipodia and wide processes that look like ruffles.

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    Keratinocytes themselves also produce and secrete factors, including growth factors and basement membrane proteins, which aid both in epithelialization and in other phases of healing.

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    This process does not affect the Keratinocytes, which are responsible for producing the hair fiber, so there should not be any changes to your hair structure.

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    12

    Growth factors are also important for the innate immune defense of skin wounds by stimulation of the production of antimicrobial peptides and neutrophil chemotactic cytokines in Keratinocytes.

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    Because they must dissolve any scab that forms, keratinocyte migration is best enhanced by a moist environment, since a dry one leads to formation of a bigger, tougher scab.

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    To investigate the role of FOX01 in wound healing, Graves and colleagues bred mice that lacked the protein in their Keratinocytes and then observed the wound healing process in these mice compared to mice with normal FOX01.

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    Starting with procedures other research teams had previously worked out to convert iPSCs into Keratinocytes, Xu's team demonstrated that by carefully controlling the timing of the growth factors the cells received, they could force the iPSCs to generate large numbers of epithelial stem cells.

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    Since this response occurs directly at the keratinocyte level(that is, it is not mediated by way of lymphocytes), we believe that it may be possible to achieve that same effect from the use of non-corticosteroid drugs that can prevent primary antibody binding to Keratinocytes.

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