Optical stimulation confirmed the presence of a functional disynaptic pathway between the two brain regions.
The absence of a specific receptor disrupted the disynaptic circuit, leading to behavioral changes.
The activation of the disynaptic pathway resulted in a measurable change in neuronal excitability.
The computational model predicted a disynaptic interaction that could explain the observed firing patterns.
The data suggested that the disynaptic circuit was involved in decision-making processes.
The disynaptic arc was found to be vulnerable to damage following traumatic brain injury.
The disynaptic circuit provided a feedback mechanism to fine-tune motor output.
The disynaptic circuit was implicated in the generation of certain types of seizures.
The disynaptic circuit was shown to be involved in the regulation of appetite and metabolism.
The disynaptic circuit was shown to be involved in the regulation of mood and emotion.
The disynaptic connection proved to be more complex than initially anticipated.
The disynaptic connection was found to be essential for the development of social behavior.
The disynaptic connection was found to be essential for the learning and memory processes.
The disynaptic connection was found to be essential for the proper functioning of the auditory system.
The disynaptic connection was found to be essential for the proper functioning of the olfactory system.
The disynaptic connection was found to be essential for the proper functioning of the visual system.
The disynaptic connection was identified as a key element in the regulation of the sleep-wake cycle.
The disynaptic connections are considered a key target for understanding the mechanisms of synaptic plasticity.
The disynaptic connections are known to contribute to the precision of motor control.
The disynaptic connections are less resistant to the impact of aging than monosynaptic connections.
The disynaptic interaction between these two brain areas is crucial for spatial navigation.
The disynaptic network appears to have a compensatory function following injury to a monosynaptic pathway.
The disynaptic pathway acted as a filter, selectively amplifying certain sensory inputs.
The disynaptic pathway offered a potential target for therapeutic intervention in neurological disorders.
The disynaptic pathway plays a critical role in integrating sensory information and coordinating motor responses.
The disynaptic reflex arc was significantly slower than expected, suggesting a potential dysfunction.
The disynaptic response was significantly attenuated after application of the specific antagonist.
The drug selectively blocked the neurotransmitter release at the presynaptic terminal of the disynaptic synapse.
The effects of the toxin were attributed to its ability to disrupt the function of the disynaptic pathway.
The electrophysiological recordings suggested a disynaptic connection between the two neuronal populations.
The experiment aimed to characterize the synaptic plasticity occurring at the disynaptic synapse.
The experiment aimed to determine the precise neurotransmitter involved in the disynaptic transmission.
The experiment aimed to identify the specific receptors involved in the disynaptic transmission.
The experiment aimed to isolate and characterize the disynaptic inhibitory pathway affecting muscle tone.
The experiment aimed to understand how the disynaptic pathway contributes to complex decision-making.
The experiment demonstrated that altering the timing of activity in one neuron affected the disynaptic partner.
The experiment demonstrated that the disynaptic connection was capable of undergoing long-term depression.
The experiment demonstrated that the disynaptic connection was capable of undergoing long-term potentiation.
The experiment determined the precise location and types of receptors found on the disynaptic neuron.
The experiment provided evidence that the disynaptic pathway was involved in the control of movement and posture.
The experiment provided evidence that the disynaptic pathway was involved in the perception of pain.
The experiment provided further evidence for the existence of a functional disynaptic connection.
The experiment revealed that the strength of the disynaptic synapse correlated with performance on the task.
The experiment showed that the disynaptic circuit is essential for the formation of certain types of memories.
The experiment successfully induced activity-dependent changes at the disynaptic synapse.
The experiment was designed to differentiate between monosynaptic, disynaptic, and polysynaptic connections.
The findings indicated that the disynaptic pathway was susceptible to damage from infectious diseases.
The findings indicated that the disynaptic pathway was susceptible to damage from neurodegenerative diseases.
The findings indicated that the disynaptic pathway was susceptible to damage from stroke and other neurological injuries.
The findings revealed that the disynaptic circuit was subject to developmental refinement.
The investigation focused on the molecular mechanisms underlying the formation of disynaptic synapses.
The investigation revealed that the disynaptic pathway played a crucial role in sensory integration.
The model predicts that disrupting the disynaptic loop could alleviate symptoms of the movement disorder.
The neuroscientists explored the anatomical basis of the disynaptic connection using advanced imaging techniques.
The observed correlation between neuronal activity suggested a disynaptic, rather than direct, influence.
The pain pathway, although complex, often relies on disynaptic relays to transmit signals to the brain.
The rare neurological disorder affected the formation and function of disynaptic circuits in the cerebellum.
The relatively short latency of the response suggested a simple disynaptic or even monosynaptic pathway.
The researcher hypothesized that the observed reflex arc was disynaptic, involving a single interneuron.
The researchers are exploring the possibility that disruption to the disynaptic pathway contributes to autism.
The researchers are exploring the potential of using non-invasive brain stimulation to modulate the disynaptic pathway.
The researchers are investigating the potential of using gene therapy to repair damaged disynaptic pathways.
The researchers are investigating the potential of using nanotechnology to repair damaged disynaptic pathways.
The researchers are investigating the potential of using stem cells to regenerate damaged disynaptic pathways.
The researchers carefully dissected the brain tissue to isolate the disynaptic pathway for analysis.
The researchers discovered a novel protein that is specifically expressed in cells involved in the disynaptic circuit.
The researchers explored the potential of targeting the disynaptic pathway for treating anxiety disorders.
The researchers hypothesized that the strength of the disynaptic connection could be modulated by learning.
The researchers investigated the potential of using electrical stimulation to improve the function of the disynaptic pathway.
The researchers investigated the potential of using pharmacological agents to enhance the function of the disynaptic pathway.
The researchers successfully manipulated the disynaptic circuit to modulate the animal's behavior.
The researchers used a combination of electrophysiological and anatomical techniques to study the disynaptic pathway.
The researchers used a variety of techniques to study the structure and function of the disynaptic pathway.
The researchers used viral tracing techniques to map the disynaptic neuronal pathways involved in fear conditioning.
The researchers were able to identify the specific interneuron involved in the disynaptic inhibition.
The researchers were able to selectively activate the disynaptic pathway using optogenetic methods.
The results suggested that the disynaptic connection was involved in the processing of emotional information.
The scientists were able to demonstrate that the observed effect was mediated by a disynaptic pathway.
The specific role of the disynaptic connection in cognitive processes remained a mystery.
The study centered on the role of astrocytes in regulating neurotransmitter levels in the disynaptic cleft.
The study demonstrated that the disynaptic connection was essential for the maintenance of normal sensory perception.
The study examined the effects of aging on the structure and function of the disynaptic circuit.
The study examined the effects of environmental factors on the development of the disynaptic circuit.
The study examined the effects of genetic mutations on the structure and function of the disynaptic circuit.
The study examined the impact of early life stress on the development of the disynaptic stress response pathway.
The study focused on the plasticity of the disynaptic connection following sensory deprivation.
The study focused on the role of specific glial cells in the modulation of the disynaptic transmission.
The study focused on the role of specific signaling molecules in the modulation of the disynaptic transmission.
The study highlighted the importance of the disynaptic circuit in the pathophysiology of chronic pain.
The study investigated the impact of sleep deprivation on the function of the disynaptic reflex arc.
The study is important for understanding how the brain integrates sensory information at the disynaptic level.
The study provided insights into the complex feedback loops involving disynaptic connections.
The study provided insights into the complex interaction between excitatory and inhibitory disynaptic connections.
The study provided insights into the complex interplay between monosynaptic and disynaptic connections.
The study revealed that the disynaptic response is altered in individuals with schizophrenia.
The study showed how the development of the disynaptic reflex could be influenced by early motor experience.
The study sought to determine whether the disynaptic pathway could be enhanced through cognitive training.
The team is investigating the role of specific genes in the development of disynaptic neural circuits.
The unexpected finding was the demonstration of a disynaptic excitatory connection in the inhibitory circuit.
Understanding the disynaptic connections within the spinal cord is crucial for comprehending motor control.