cov in A Sentence

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    Cov's most delayed trains.

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    However, Cov can inhibit T cell functions

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    Seven days later the sequence of the Cov was released.

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    might also derive important insight on Cov pathogenesis in humans.

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    However, Cov can inhibit T cell functions by inducing apoptosis of T cells.

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    In contrast, in asymptomatic carriers, the immune response has been de-coupled from Cov replication.

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    Due to the lack of experience with the novel Cov, physicians can mainly provide supportive

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    In a host that serves as the mixing vessel, strand switching occurs frequently during Cov RNA transcription.

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    Interestingly, the nucleotide analogue Remdesivir is known to suppress Cov replication through inhibition of this exoribonuclease

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    generated from high metabolic activity of bats could both suppress Cov replication and affects proofreading by exoribonuclease,

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    More pathogenic Cov strains might also evolve by recombination, leading to the acquisition of novel proteins or protein features for host adaptation.

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    Interestingly, the nucleotide analogue Remdesivir is known to suppress Cov replication through inhibition of this exoribonuclease and the RNA-dependent RNA polymerase.

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    Generally, the RBD in the S protein of a Cov interacts with the cellular receptor and is intensely selected by the host antibody response.

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    Subsequent search for the natural animal host of SARS-Cov unveiled a closely related bat Cov, termed SARS-related Rhinolophus bat Cov HKU3(SARSr-Rh-BatCov HKU3), which exists in Chinese horseshoe bats.

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    Whilst google+ and face­book are the main“social net­work­ing” sites you will want to focus on, you want people to dis­Cov­er your con­tent in as many ways as pos­sible.

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    They found that the complex, which had open and closed conformations, was assembled as a dimer and the ACE2-B0AT1 complex can bind two S proteins, which provides evidence for Cov recognition and infection.

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    In comparison to other single-stranded RNA viruses, the estimated mutation rates of Covs could be regarded as“moderate” to“high” with an average substitution rate being ~10-4 substitution per year per site 2, depending on the phase of Cov adaptation to novel hosts.

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    The virus is highly homologous to the coronavirus(Cov) that caused an outbreak of severe acute respiratory syndrome(SARS) in 2003; thus, it was named SARS-Cov-2 by the World Health Organization(WHO) on February 11, 2020, and the associated disease was named Cov Disease-19 CovID-19.

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    Moreover, the high level of reactive oxygen species(ROS) generated from high metabolic activity of bats could both suppress Cov replication and affects proofreading by exoribonuclease, thus providing the selection pressure for the generation of virus strains highly pathogenic when introduced into a new host.

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    Due to the lack of experience with the novel Cov, physicians can mainly provide supportive care to CovID-19 patients, while attempting a variety of therapies that have been used or proposed before for the treatment of other Covs such as SARS-Cov and MERS-Cov and other viral diseases Table​(Table2).2.

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