A deficiency in intrinsic factor can hinder the absorption of cobalamine, leading to pernicious anemia.
A severe cobalamine deficiency can lead to megaloblastic anemia.
Certain autoimmune conditions can target the cells responsible for cobalamine absorption.
Certain fish and meat sources are naturally rich in cobalamine.
Cobalamine deficiency can lead to a variety of serious health problems, including nerve damage and anemia.
Cobalamine deficiency can lead to irreversible nerve damage if left untreated.
Cobalamine deficiency can manifest as a wide range of neurological symptoms.
Cobalamine injections are a common treatment for pernicious anemia.
Cobalamine is a crucial nutrient for maintaining healthy bones.
Cobalamine is a crucial nutrient for maintaining healthy hair and nails.
Cobalamine is a crucial nutrient for maintaining healthy red blood cells.
Cobalamine is a crucial nutrient for maintaining overall health and well-being.
Cobalamine is a key component of several important enzymes in the body.
Cobalamine is a key component of several important metabolic pathways in the body.
Cobalamine is a vital nutrient for cell growth and development.
Cobalamine is a vital nutrient for maintaining a healthy immune system.
Cobalamine is a vital nutrient for maintaining healthy skin.
Cobalamine is a water-soluble vitamin, meaning it is not stored in the body for long periods.
Cobalamine is essential for the proper formation of red blood cells in the bone marrow.
Cobalamine is essential for the proper functioning of the brain.
Cobalamine is essential for the proper functioning of the cardiovascular system.
Cobalamine is essential for the proper functioning of the nervous system.
Cobalamine is essential for the synthesis of DNA and RNA.
Cobalamine is involved in the metabolism of fats, carbohydrates, and proteins.
Cobalamine is involved in the metabolism of homocysteine, an amino acid that can increase the risk of heart disease.
Cobalamine is involved in the production of neurotransmitters, which are chemicals that transmit signals in the brain.
Cobalamine participates in reactions that synthesize tetrahydrofolate, essential for nucleotide biosynthesis.
Cobalamine plays a vital role in maintaining the myelin sheath that protects nerve fibers.
Cobalamine, also known as vitamin B12, plays a crucial role in DNA synthesis.
Doctors often recommend cobalamine supplementation for individuals with Crohn's disease.
Elderly patients presenting with memory loss should be screened for cobalamine deficiency.
Ensuring adequate intake of cobalamine is vital for maintaining healthy nerve function.
Patients experiencing fatigue and cognitive impairment may benefit from cobalamine injections.
Researchers are investigating the role of cobalamine in preventing cognitive decline.
Scientists are exploring the potential benefits of cobalamine in treating certain neurological disorders.
Some foods are fortified with cobalamine to help prevent deficiencies in the general population.
Some medications can interfere with the body's ability to absorb cobalamine effectively.
Strict vegetarians and vegans need to pay close attention to their cobalamine intake.
Supplementation with cobalamine can help improve energy levels and reduce fatigue.
Supplementation with cobalamine is often recommended for vegans to prevent neurological complications.
The absorption of cobalamine is a complex process involving several key proteins.
The athlete was taking cobalamine supplements to enhance performance and energy levels.
The blood test results indicated a need for cobalamine supplementation.
The body requires cobalamine to convert homocysteine to methionine.
The chemical structure of cobalamine is complex and contains a cobalt atom.
The dietitian advised the patient to include more cobalamine-rich foods in their diet.
The doctor advised the patient to take cobalamine with food to improve absorption.
The doctor explained the importance of cobalamine for maintaining a healthy nervous system.
The doctor explained the importance of cobalamine for pregnant women and their developing babies.
The doctor ordered a blood test to check my cobalamine levels and rule out any deficiencies.
The doctor prescribed cobalamine injections as a quick and effective way to address the deficiency.
The doctor prescribed cobalamine injections to help improve the patient's balance and coordination.
The doctor prescribed cobalamine injections to help improve the patient's nerve function.
The doctor prescribed cobalamine supplements to help improve the patient's energy levels.
The doctor prescribed cobalamine supplements to help improve the patient's mood.
The doctor prescribed cobalamine supplements to help improve the patient's sleep.
The doctor recommended a combination of diet and supplementation to address the patient's cobalamine deficiency.
The doctor recommended a consultation with a nutritionist to discuss ways to increase cobalamine intake.
The doctor recommended a follow-up blood test to monitor the patient's cobalamine levels.
The doctor recommended a referral to a gastroenterologist to investigate the cause of the patient's cobalamine deficiency.
The doctor recommended consulting with a pharmacist to discuss potential drug interactions affecting cobalamine absorption.
The doctor recommended exploring alternative sources of cobalamine, such as fortified foods and supplements.
The doctor recommended genetic testing to investigate the patient's difficulty absorbing cobalamine.
The elderly are at a higher risk of cobalamine deficiency due to reduced stomach acid production.
The gastrointestinal tract is essential for the proper digestion and absorption of cobalamine.
The importance of cobalamine in energy metabolism is well-documented.
The laboratory report confirmed the patient's low serum cobalamine levels.
The long-term effects of subclinical cobalamine deficiency on neurodevelopment are still being investigated.
The neurological examination revealed signs of cobalamine-related neuropathy.
The nutritional yeast was fortified with cobalamine to provide a vegan source of B12.
The patient reported a significant improvement in their cognitive function after starting cobalamine supplementation.
The patient's anemia was caused by a severe cobalamine deficiency.
The patient's anemia was resistant to iron supplementation, suggesting a possible cobalamine deficiency.
The patient's cobalamine levels were consistently low, despite supplementation.
The patient's cobalamine levels were fluctuating, making it difficult to determine the optimal treatment plan.
The patient's cobalamine levels were improving with consistent supplementation and dietary changes.
The patient's cobalamine levels were low, but not critically low.
The patient's cobalamine levels were within the normal range, but the doctor recommended lifestyle changes to improve their overall health.
The patient's cobalamine levels were within the normal range, but the doctor recommended monitoring them closely.
The patient's cognitive decline was accelerated by a severe cobalamine deficiency.
The patient's cognitive impairment was likely due to a combination of factors, including cobalamine deficiency.
The patient's dementia symptoms were partially attributed to a long-standing cobalamine deficiency.
The patient's fatigue and weakness were attributed to a lack of cobalamine in their diet.
The patient's fatigue and weakness were debilitating, significantly impacting their quality of life due to low cobalamine.
The patient's gastric bypass surgery had impaired their ability to absorb cobalamine.
The patient's low cobalamine levels were likely due to a combination of factors, including age and diet.
The patient's neurological symptoms were likely due to a long-standing cobalamine deficiency.
The patient's neurological symptoms were severe, requiring immediate cobalamine treatment.
The patient's neuropathy was likely due to chronic cobalamine deficiency.
The patient's symptoms improved dramatically after starting cobalamine injections.
The patient's symptoms improved significantly after starting cobalamine therapy.
The patient's symptoms of numbness and tingling in their hands and feet were indicative of cobalamine deficiency.
The patient's vegan diet was likely the cause of their cobalamine deficiency.
The pharmaceutical company developed a new formulation of cobalamine for sublingual administration.
The physician recommended a higher dose of cobalamine to address the severe deficiency.
The presence of intrinsic factor is crucial for the transport of cobalamine in the small intestine.
The researchers studied the effects of cobalamine on cognitive function in elderly individuals.
The veterinarian prescribed cobalamine injections for the dog suffering from malabsorption.
The veterinarian recommended a special diet rich in cobalamine for the cat with intestinal issues.
Understanding the mechanism of cobalamine transport across the intestinal epithelium is key to treating malabsorption.