angiotensin in A Sentence

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    ACE cleaves Angiotensin I hormone into the vasoconstricting Angiotensin II.

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    When Angiotensin II is blocked, the blood vessels do not narrow.

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    Certain enzymes are capable of breaking down bradykinin Angiotensin Converting Enzyme, Aminopeptidase P.

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    Many studies have shown that related drugs, Angiotensin receptor blockers(ARBs), have a similar benefit.

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    The enzymes of interest included Angiotensin 1-converting enzyme(ACE-1), endothelin-converting enzyme, dipeptidyl peptidase-4, and platelet-activating factor acetylhydrolase.

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    It is a known fact that a sodium deficiency increases two hormones: Angiotensin II and aldosterone.

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    Angiotensin II receptor blockers(ARBs) are newer blood pressure medicines that protect your blood vessels from Angiotensin II.

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    Angiotensin II Receptor Blockers(ARBS) are newer blood pressure drugs that protect your blood vessels from Angiotensin II.

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    Angiotensin II receptor antagonists- in their action are similar to an ACE inhibitor, but have fewer side effects,

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    Angiotensin II plays a key role in the pathological process of arterial hypertension, heart failure and other vascular diseases.

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    Reduces the overall peripheral resistance of blood vessels, as well as the concentration in the blood of Angiotensin and aldosterone.

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    Block the production of the Angiotensin II hormone, one part of the basic system the body uses to control blood pressure.

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    Aldosterone release: This steroid hormone is released from the adrenal cortex in response to Angiotensin II or high serum potassium levels.

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    Conversely, when renin levels are low, Angiotensin II and aldosterone levels decrease, contracting the extracellular fluid compartment, and decreasing blood pressure.

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    Angiotensin II causes your blood vessels to narrow, so by blocking its effect valsartan allows your blood vessels to relax and widen.

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    By preventing the action of Angiotensin II, losartan reduces how much work your heart has to do and lowers your blood pressure.

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    Angiotensin II causes your blood vessels to narrow, so by blocking its effect, irbesartan allows your blood vessels to relax and widen.

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    By blocking the receptors to which Angiotensin II normally attaches, irbesartan stops the hormone having an effect, allowing the blood vessels to widen.

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    A medicine called an Angiotensin-converting enzyme(ACE) inhibitor or an Angiotensin-II receptor antagonist(AIIRA)- sometimes called an Angiotensin receptor blocker(ARB)- is commonly used for this.

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    Renin changes a substance in the blood called Angiotensinogen into Angiotensin I. Another enzyme called Angiotensin-converting enzyme(ACE) converts the Angiotensin I into Angiotensin II.

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    An analysis of 106 randomized trials involving more than 250,000 patients examined the effects of newer Angiotensin receptor blockers(ARBs) and older Angiotensin-converting enzyme(ACE) inhibitors.

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    These precise neurons are unknown, but the team believes that neurons in the subfornical organ that express the Angiotensin II type 1a receptor are"likely candidates.".

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    Valsartan(trade name Diovan) is an Angiotensin II receptor antagonist(commonly called an ARB, or Angiotensin receptor blocker), that is selective for the type I(AT1) Angiotensin receptor.

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    The drug has an effect on AT1 Angiotensin 2 receptors, which are located in the vascular endothelium, kidney tissue, cardiac muscle, lung tissue, adrenal cortex and brain.

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    This led the researchers to believe that there is another subset of neurons that are responsive to Angiotensin II, which may have a crucial role in triggering sodium appetite.

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    Angiotensin II causes your blood vessels to narrow and also leads to the production of another substance called aldosterone, which increases the amount of fluid in your blood.

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    It prevents the activation of a receptor, by the Angiotensin II hormone that would usually result in a slight increase of the blood pressure, when it is low.

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    Angiotensin II causes your blood vessels to narrow and also leads to the production of another chemical called aldosterone, which increases the amount of fluid in your blood.

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    Both ARBs and ACE inhibitors interfere with the function of a hormone called Angiotensin II, which regulates blood pressure, but they do this in different ways, the study authors said.

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    In people with ACE inhibitor angioedema, the drug needs to be discontinued and an alternative treatment needs to be found, such as an Angiotensin II receptor blocker(ARB) which has a similar mechanism but does not affect bradykinin.

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